β3-adrenoceptor, glutathionylation, and diabetic cardiomyopathy. Focus on “β3-adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade”

Stephen M. Black

Research output: Contribution to journalEditorial

3 Citations (Scopus)
Original languageEnglish (US)
Pages (from-to)C283-C285
JournalAmerican Journal of Physiology - Cell Physiology
Volume309
Issue number5
DOIs
StatePublished - Jan 1 2015

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Adrenergic beta-3 Receptors
Diabetic Cardiomyopathies
Sodium-Potassium-Exchanging ATPase
Adrenergic beta-Agonists
Insulin Receptor
Drug Delivery Systems
Hyperglycemia
Adrenergic Receptors
Glutathione
Oxidative Stress

ASJC Scopus subject areas

  • Physiology
  • Cell Biology

Cite this

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title = "β3-adrenoceptor, glutathionylation, and diabetic cardiomyopathy. Focus on “β3-adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade”",
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JF - American Journal of Physiology - Heart and Circulatory Physiology

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