Increased long chain acyl CoA in duchenne muscular dystrophy

James E. Carroll; Aida Villadiego; Michael H. Brooke

doi:10.1212/wnl.33.11.1507

Increased long chain acyl CoA in duchenne muscular dystrophy

James E. Carroll, Aida Villadiego, Michael H. Brooke

Research output: Contribution to journal › Article › peer-review

17 Scopus citations

Abstract

Compared with normal or denervated human muscle, long chain acyl CoA was increased in muscle from patients with Duchenne dystrophy. Free and short chain acylcarnitine were reduced in Duchenne muscle, whereas long chain acylcarnitine was preserved. The accumulation of long chain fatty acid derivatives may imply disruption of fatty acid oxidation.

Original language	English (US)
Pages (from-to)	1507-1510
Number of pages	4
Journal	Neurology
Volume	33
Issue number	11
DOIs	https://doi.org/10.1212/wnl.33.11.1507
State	Published - Nov 1983
Externally published	Yes

ASJC Scopus subject areas

Clinical Neurology

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title = "Increased long chain acyl CoA in duchenne muscular dystrophy",

abstract = "Compared with normal or denervated human muscle, long chain acyl CoA was increased in muscle from patients with Duchenne dystrophy. Free and short chain acylcarnitine were reduced in Duchenne muscle, whereas long chain acylcarnitine was preserved. The accumulation of long chain fatty acid derivatives may imply disruption of fatty acid oxidation.",

author = "Carroll, {James E.} and Aida Villadiego and Brooke, {Michael H.}",

year = "1983",

month = nov,

doi = "10.1212/wnl.33.11.1507",

language = "English (US)",

volume = "33",

pages = "1507--1510",

journal = "Neurology",

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publisher = "Lippincott Williams and Wilkins",

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TY - JOUR

T1 - Increased long chain acyl CoA in duchenne muscular dystrophy

AU - Carroll, James E.

AU - Villadiego, Aida

AU - Brooke, Michael H.

PY - 1983/11

Y1 - 1983/11

N2 - Compared with normal or denervated human muscle, long chain acyl CoA was increased in muscle from patients with Duchenne dystrophy. Free and short chain acylcarnitine were reduced in Duchenne muscle, whereas long chain acylcarnitine was preserved. The accumulation of long chain fatty acid derivatives may imply disruption of fatty acid oxidation.

AB - Compared with normal or denervated human muscle, long chain acyl CoA was increased in muscle from patients with Duchenne dystrophy. Free and short chain acylcarnitine were reduced in Duchenne muscle, whereas long chain acylcarnitine was preserved. The accumulation of long chain fatty acid derivatives may imply disruption of fatty acid oxidation.

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DO - 10.1212/wnl.33.11.1507

M3 - Article

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AN - SCOPUS:0021085090

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SP - 1507

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JO - Neurology

JF - Neurology

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ER -

Increased long chain acyl CoA in duchenne muscular dystrophy

Abstract

ASJC Scopus subject areas

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