Defective [U-14 C] palmitic acid oxidation in duchenne muscular dystrophy

James Edwin Carroll, Beverly J. Norris, Michael H. Brooke

Research output: Contribution to journalArticle

18 Citations (Scopus)

Abstract

Compared with normal skeletal muscle, muscle from patients with Duchenne dystrophy had decreased [U-14 C] palmitic acid oxidation. [l-14 C] palmitic acid oxidation was normal. These results may indicate a defect in intramitochondrial fatty acid oxidation.

Original languageEnglish (US)
Pages (from-to)96-97
Number of pages2
JournalNeurology
Volume35
Issue number1
StatePublished - Jan 1 1985
Externally publishedYes

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Duchenne Muscular Dystrophy
Palmitic Acid
Skeletal Muscle
Fatty Acids
Muscles

ASJC Scopus subject areas

  • Clinical Neurology

Cite this

Defective [U-14 C] palmitic acid oxidation in duchenne muscular dystrophy. / Carroll, James Edwin; Norris, Beverly J.; Brooke, Michael H.

In: Neurology, Vol. 35, No. 1, 01.01.1985, p. 96-97.

Research output: Contribution to journalArticle

Carroll, JE, Norris, BJ & Brooke, MH 1985, 'Defective [U-14 C] palmitic acid oxidation in duchenne muscular dystrophy', Neurology, vol. 35, no. 1, pp. 96-97.
Carroll, James Edwin ; Norris, Beverly J. ; Brooke, Michael H. / Defective [U-14 C] palmitic acid oxidation in duchenne muscular dystrophy. In: Neurology. 1985 ; Vol. 35, No. 1. pp. 96-97.
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